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製造業轉型之研究－以TS自動門為例

為了解決TME stock的問題，作者洪國彰 這樣論述：

現有的文獻當中，已經有許多研究探討在現今的紅海市場中，製造業該如何轉型，儘管有許多服務化相關的文獻，卻很少有文獻探討具體的服務化過程與方法。因此，本研究深入探討製造業如何透過服務化轉型及其對公司的影響，期望未來能將研發的策略應用於TS公司的管理系統。 本研究是一個質化之個案研究，個案公司為TS自動門公司，主要的客群為連鎖便利商店與超市。客戶始終是企業最重要的資源之一，以客戶為中心不僅可以提高顧客忠誠度，亦能在與顧客互動的過程中，了解複雜的顧客需求。本研究以客戶為中心，探討客戶需求的複雜性與製造商如何從商品提供者成為服務提供者的過程。 研究發現服務化可以使企業滿足日益複雜的

客戶需求，且服務收益高於產品收益，不僅可以增加公司的收益，亦能增加客戶的忠誠度。此外，當製造商的策略從以商品與技術為中心轉變為以客戶為中心，製造商非但提高了將技術與客戶價值相結合的能力，同時也提升了自身在產業中的競爭力。本研究探討在服務化的過程中，個案公司如何調整公司內部制度與運用策略，成功地將TS自動門公司轉型。

以小鼠模型研究自然殺手細胞癌症免疫治療

為了解決TME stock的問題，作者黃世雯 這樣論述：

ContentsContents ....................................................................................................................................... IFigure contents ......................................................................................................................... IIITabl

e contents .......................................................................................................................... IVAbbreviations ............................................................................................................................ V中文摘要 ...................

.............................................................................................................. VIIAbstract .................................................................................................................................... IXChapter 1. Introduction ..................

............................................................................................ 11.1 Natural killer (NK) cells .............................................................................................. 11.1.1 Development of NK cells ..................................................

................................... 11.1.2 Recognition of tumor cells by NK cells and NK cell activation .......................... 21.1.3 Anti-tumor function of NK cells .......................................................................... 41.2 The immunosuppressive tumor microenvironment (TME

) ......................................... 61.2.1 The immunosuppressive molecules produced by tumor cells .............................. 61.2.2 Recruitment and accumulation of suppressive immune cells .............................. 81.3 Autologous NK cell therapy in cancer .........................

............................................... 91.4 Cyclophosphamide .................................................................................................... 101.5 The EO771 breast cancer model ................................................................................ 111.6 Hypoth

esis ................................................................................................................. 12Chapter 2. Materials and Methods ........................................................................................... 132.1 Mice ...........................................

..................................................................................... 132.2 Preparation of single cell suspension from the BM, spleen, liver and lung .............. 132.3 Ex vivo expansion of NK cells ..................................................................................

142.4 Tumor cell lines ......................................................................................................... 142.5 EO771 tumor models ................................................................................................. 152.6 Analysis of lung metastasis ..............

......................................................................... 16II2.7 Flow cytometry and sorting ....................................................................................... 162.8 In vitro NK-cell cytotoxicity assay .............................................................

............... 172.9 NK cell trafficking analysis ....................................................................................... 172.10 In vivo T cell depletion .............................................................................................. 172.11 Statistical analysis ......

................................................................................................ 18Chapter 3. Results .................................................................................................................... 193.1 Characterization of the ex vivo expanded murine NK cells ..

.................................... 193.2 The EO771 tumor-resected mice represent a metastasis model ................................ 203.3 Adoptive syngeneic NK cell therapy is effective in EO771-resected mice withrelatively low tumor burden ........................................................

......................................... 213.4 T cells are essential for the efficacy of NK cell therapy in EO771-resected mice .... 223.5 Adoptive syngeneic NK cell therapy is not effective in EO771-bearing mice ......... 243.6 NK cells and CTX show synergistic therapeutic effect on EO771 tumor mo

dels .... 25Chapter 4. Discussion ............................................................................................................... 284.1 Comparison of ex vivo expanded NK cells with ILC1, conventional NK (cNK) cellsand memory NK cells ..................................................

......................................................... 284.2 Autologous NK cell therapy is likely beneficial for treating cancer of relative lowtumor burden ......................................................................................................................... 284.3 The mechanis

m of NK cell therapy ........................................................................... 304.4 The mechanism of CTX/NK-cell therapy. ................................................................ 324.5 Other rational therapy combinations ....................................................

..................... 334.6 Prospectives ............................................................................................................... 34Chapter 5. Conclusion .............................................................................................................. 35Chapter 6.

References .............................................................................................................. 36